Would hepatic gluconeogenesi only help with patients with diabetes type 2?

No, ↑es the Problems: With healthy metabolism, ~50% of more of glucose is produced by hepatic gluconeogenesis (sugar production from internal protein & fat) & prevents low bg fasting. However, in type ii, glucagon & gluconeogenesis increase after eating, driving blood sugar levels even higher. Type ii is excess sugar, intake & production, not too little, the reason bg is routinely high. As dm worsens problem ↑es.