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A member asked:
Why does primary hypoaldosteronism lead to increased renin and angiotensin ii, but secondary hypoaldosteroninim does not?
A doctor has provided 1 answer
Renin-AT II-ALD axis: With primary (1°) hypoaldosteronism, feedback on the renin-angiotensin (AT) II-aldosterone (ALD) axis would cause the kidneys to increase renin and thereby AT II to try ^ adrenal ALD. In 2° hypoaldo, there is usually kidney damage (for example, due to diabetes) which decreases renin and hence AT II (which is produced in response to renin). This causes ALD (which depends on AT II) to v (hence 2°).
Answered 8/18/2014
3.8k views
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