Question 1

will epinephrine increase cardiac output?

Accepted Answer

Yes: It makes the heart contract more forcefully and makes it beat faster. Both effects will increase the cardiac output.

Question 2

can beta blockers cause cardiac arrest?

Accepted Answer

Yes: An overdose in beta blocker can cause cardio respiratory arrest -- only take beta blocker under a doctor supervision and direction.

Question 3

will acetylcholine raise cardiac output?

Accepted Answer

Not expected to: Acetylcholine (parasympathetic nervous system) should have the opposite effect from Epinephrine (sympathetic nervous system) and therefore should slow the heart rate and decrease the force of contraction of the heart. Both effects would tend to lower the cardiac output (stroke volume times the heart rate). The stroke volume is the amount of blood ejected with each heart beat.

Question 4

hyperkalemia causes cardiac arrest in systole?

Accepted Answer

No: Hyperkalemia causes cardiac arrest in diastole.

Question 5

are these drugs used to reduce cardiac output: calcium channel blockers and beta (receptor) blockers?

Accepted Answer

No: They are used to slow the heart rate. Can be used with fast heart rate problems, but can also aide in blood pressure. Usually will increase cardiac output.

Question 6

can beta blocker prevent vf or sudden cardiac arrest?

Accepted Answer

Yes: Beta blockers are commonly used to decrease the frequency and severity of ventricular arrhythmias.  Depending on your situation (state of your heart, e.t.c.), more testing, different medication, or even a defibrillator may be needed.

Question 7

why is digoxin contraindicated in ventricular tachycardias?

Accepted Answer

Ventricular tachycar: Ventricular tachycardia is a dangerous cardiac rhythm and one of the abnormal rhythms caused by digoxin toxicity. Digoxin could cause worsening of VT or conversion to vf.

Question 8

benefits of the selective beta blocker toprol (metoprolol) for heart failure/ cardiomyopathy?

Accepted Answer

Reverses Ht Failure: Toprol xl (specifically that formulation, not metoprolol tartrate) and Carvedilol (generic or Coreg (carvedilol) brand name) have been shown in large, well controlled clinical trials to slow and sometimes reverse the damage of heart failure. They operate by blocking a malfunctioning feedback loop of hormones and signaling molecules, ultimately by blocking the effect of adrenaline on the heart. Good question!

Question 9

how do beta-blockers increase cardiac output?

Accepted Answer

They don't: Cardiac output is the product of heart rate and stroke volume. Beta blockers decrease heart rate and the strength of contraction (contractility). Thus, beta blockers would not be expected to increase cardiac output.

Question 10

do selective adeno-adenergic blockers (prazosin, terazosin, doxazosin, trimazosin) protect from acute cocaine-induced vasoconstrictive damage?

Accepted Answer

Some: Cocaine causes it's effect by preventing the reuptake of monamine (epinephrine, etc) and causes these neurotransmitters to continue exciting the a1 receptor.  The meds listed block alpha-1 receptors, so theoretically will blunt the effects of Cocaine on some vessels, but focus mostly (esp. Tamsulosin) on a specific sub-type (alpha-1a) that is mostly in the bladder, not in the vessels.

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A member asked:

Will epinephrine increase cardiac output?

A member asked:

Can beta blockers cause cardiac arrest?

A member asked:

Will acetylcholine raise cardiac output?

A member asked:

Hyperkalemia causes cardiac arrest in systole?

A member asked:

Are these drugs used to reduce cardiac output: calcium channel blockers and beta (receptor) blockers?

A member asked:

Can beta blocker prevent vf or sudden cardiac arrest?

A member asked:

Why is digoxin contraindicated in ventricular tachycardias?

A member asked:

Benefits of the selective beta blocker toprol (metoprolol) for heart failure/ cardiomyopathy?

A member asked:

How do beta-blockers increase cardiac output?

A member asked:

Do selective adeno-adenergic blockers (prazosin, terazosin, doxazosin, trimazosin) protect from acute cocaine-induced vasoconstrictive damage?

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