Related Questions

Can joint hypermobility be dangerous?

Probably not. Isolated joint hypermobility is not dangerous. When part of a syndrome like marfan's syndrome, there may be associated problems that are more serious. Marfan syndrome has a range of expressions, from mild to severe. The most serious complications are defects of the heart valves and aorta. It may also affect the lungs, the eyes, the dural sac surrounding the spinal cord, and the skeleton.
Not common form. Theory that uterus-lining tissue migrates to layer of tissue (peritoneum) lining the pelvic cavity & shedding blood at each menses.  alternative theory is hormone "relaxin" loosens pelvic ligaments already loosened by joint hypermobility syndrome, multiple childbirths or injury; causing tendonitis of transversus abdominis muscle at anterior superior iliac crests.  see my board quora. Com.

How do I know if have joint hypermobility?

Fingers bend back. Hypermobility is diagnosed when several joints extend (move backwards) more than they should, elbows small knucles and knees are common. It is not serious generally, and found in many gymnasts and indian rubber circus people.
Beighton scale. If pain is present in the setting of hypermobility, most physicians will utilize the Brighton Criteria for diagnosis of JHS. If an individual has no pain then a Beighton score = or > 5/6 is sufficient for hypermobility alone.

What is the definition or description of: Joint hypermobility?

Laxity. There is a normal range of motion that most joints have as they are moved. Joint hyper mobility describes a range of motion in a particular joint that is more than normal. Hyper mobile joints are at increased risk of subluxations and dislocations. The term ligamentous laxity implies joint hyper mobility.

What is joint hypermobility syndrome?

See below. Hypermobile joints are joints that move beyond the normal range of motion. Joints most commonly affected are the elbows, wrists, fingers, and knees. Hypermobile joints often occur in otherwise healthy and normal children. This is called benign hypermobility syndrome.
Painful Joints. Joints that exceed their normal range of motion are "hypermobile". The condition is found mostly in women and has genetic relationships. There is an association of joint hypermobility and #fibromyalgia. Hypermobile joints potentiate people who have this condition to mechanical injury.

What is joint hypermobility syndrome?

Fingers bend back. Hypermobility is diagnosed when several joints extend (move backwards) more than they should, elbows small knucles and knees are common. It is not serious generally, and found in many gymnasts and indian rubber circus people.

What can I do about joint hypermobility syndrome?

Strong joint muscles. Joint stability is largely derived from the presence of passive stabilizers such as the ligaments and capsule around the joint that limit joint motion in all the possible directions. The dynamic stabilizers are the muscles surrounding each joint that can be helped by making them stronger and more efficient at helping to stabillize the joints in question. Physical therapy helps strengthen muscles.
Exercise. Evidence on effective treatments in JHS is variable but overall it seems that a program focused on increasing aerobic capacity, maintaining good dynamic control, and improving proprioception tend to be most successful. Meeting with a physical therapist who can help put together a safe and effective program would be ideal.

Does anyone have any info on joint hypermobility syndrome?

Ehlers-danlos syndro. There is much info on these--more on the famous ehlers-danlos syndrome. Follow this link: http://www. Ncbi. Nlm. Nih. Gov/pubmedhealth/pmh0002439/ hope you find the answer your are looking for. Good luck.
Poorly understood dx. JHS is symptomatic hypermobility. Many individuals who have hypermobility never have pain or require any treatment. When hypermobility is found associated with pain, or a few other criteria (see Brighton criteria) it is termed JHS. It is presumed to be caused by a defect in collagen but rarely have collagen related genes been implicated (TNXB gene).
The Brits. The best clinical medical literature on the subject seems to originate from England, where there are over two dozen clinics devoted to this disorder, which is a spectrum disorder from "benign" form to EDS and Marfans, both severe manifestations. IMO, the familial nature of fibromyalgia is explained by the marked association of fibromyalgia and JHS, which has genetic underpinnings.