Doctor insights on:
Mobitz Type 1 Heart Block
Mobitz I : Second degree mobitz i av block occurs when the pr interval gradually lengthens followed by a non-conducted p-wave. ...Read more
Mixed Terms: Your question is contradictory. Mobitz i is wenckebach. I suspect you meant 2nd degree heart block. In which case, yes, stimulants increase the overall heart rate, increasing the stress on the lower conduction system we call the his-purkinje system, and increasing the likelihood that it will block. If the problem improves with caffeine, the problem was likely at the av nodal level instead. ...Read more
Degree: Mobitz1: the amount of time it takes to traverse the av node (pr interval) increases until there is failure of an impuse and beat which then resets the timing. Mobitz2: there is unpredictable complete failure to activate the ventricles, often in the his-purkinje system. ...Read more
What is atrial flutter with variable AV block with premature ventricular or aberrantly conducted complexes? is same with AV Block heart degres 1?
Not the same: Both are types of fatigue in the heart's wiring system. 1st degree heart block is when there is a delay in the signal getting from atria down to ventricles. That's minor. In more advanced heart block, some beats dont get through at all. In lbbb, there is no block between atria + ventricles but there is a delay in the signal spreading within the ventricles, which doesnt cause symptoms by itself. ...Read moreSee 1 more doctor answer
Predominantly sinus rhythm, with evidence of 2:1AV heart block (type II). 3rd degree block reported, now thought to be 2 beat P wave asystole. worry?
Can a paroxysmal atrial fibrillation and av node reentry tachycardia increase the risk for a heart attack?
Could there be a connection between my mobitz 1 heart block and the slight heart murmur the cardiologist heard?
My recent EKG reads sinus brady with 1st degree AV Block incomplete right bundle branch block. Am I at a greater risk for heart failure or a fib?
Correct: AllButBrady: damaged muscle cells, most commonly from blood supply issues (artery disease, by US autopsy studies >8 decades, present 1/2 population by age 7), arteries enlarge, not narrow, thus not clinically apparent for decades until vulnerable plaques rupture releasing WBCs triggering clots & blood flow blocked to capillaries. Same issue underlying atrial fib. All issues missed by conventional tests. ...Read moreSee 1 more doctor answer
Please can you explain what atrial flutter with variable av block
compatible with a bundle branch block. Possible inferior infarction, probably old is?
ECG interpretation.: Atrial flutter: your heart rhythm is abnormal (the atria are contracting very rapidly); variable block: there are a variable number of atrial contractions for every ventricular contraction. A bundle branch block: the ECG complex representing each ventricular contraction is widened due to a block of one branch of your heart's conduction system. Q waves on the ECG suggest an infarct (? Age). ...Read moreSee 2 more doctor answers
Results of a 24-hour holter test: 9 VPC's, 381 APC's, 3 nonsustained atrial tachycardia 4-9 beats each, at 100-102 beats/min. Is this normal?
LA enlargement : La enlargement is likely due to one of several causes including high blood pressure, valve disease or other structural heart problems. This could be related to the first degree heart block since if the atria is big then it may take longer to conduct from the atria to the ventricle. The best way to evaluate la or left atrial size is by an ultrasound or echocardiogram of the heart. ...Read moreSee 1 more doctor answer
SVT Is the lifetime chance of devoloping venticular fibrillation due to accesory pathway capable of anterograde conduction <1% When ecg was normal?
Wpw : Extremely low if antegrade conduction so rare ...Read more
Is treatment required for symptomatic bradycardia, heart rate 35, with bigeminal PVCs, normal blood pressure, no history of heart disease?
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