University of colorado denver endocrinology
Avista adventist hospital
Boulder community hospital
Childrens hospital colorado
Denver health and hospital
Exempla lutheran medical center
Exempla st joseph hospital
Littleton adventist hospital
Longmont united hospital
North suburban medical center
Rose medical center
Sky ridge medical center
St anthony hospital
St anthony north hospital
Swedish medical center
University of colorado hospital
Airway tissue factor-dependent coagulation activity in response to sulfur mustard analog 2-chloroethyl ethyl sulfide.
Am. J. Physiol. Lung Cell Mol. Physiol., Jan;302(1):L82-92 (2012)
Interaction and localization of synthetic nanoparticles in healthy and cystic fibrosis airway epithelial cells: effect of ozone exposure.
J Aerosol Med Pulm Drug Deliv., Feb;25(1):7-15 (2012)
Adenosine A2A receptor is a unique angiogenic target of HIF-2alpha in pulmonary endothelial cells.
Proc. Natl. Acad. Sci. U.S.A., Jun;106(26):10684-9 (2009)
Treatment with the catalytic metalloporphyrin AEOL 10150 reduces inflammation and oxidative stress due to inhalation of the sulfur mustard analog 2-chloroethyl ethyl sulfide.
Free Radic. Biol. Med., May;48(9):1188-96 (2010)
Chlorine gas inhalation: human clinical evidence of toxicity and experience in animal models.
Proc Am Thorac Soc., Jul;7(4):257-63 (2010)
Extracellular adenosine triphosphate: a potential regulator of vasa vasorum neovascularization in hypoxia-induced pulmonary vascular remodeling.
Chest., Dec;128(6 Suppl):608S-610S (2005)
Activation of a novel isoform of methionine adenosyl transferase 2A and increased S-adenosylmethionine turnover in lung epithelial cells exposed to hyperoxia.
Free Radic. Biol. Med., Jan;40(2):348-58 (2006)
Cholesterol interferes with the MTT assay in human epithelial-like (A549) and endothelial (HLMVE and HCAE) cells.
Int. J. Toxicol., 25(1):17-23 (2006)
Commentary on "Hypoxia, hypoxic signaling, tissue damage, and detection of reactive oxygen species (ROS)".
Free Radic. Biol. Med., Mar;40(6):923-7 (2006)
Endothelial Akt activation by hyperoxia: role in cell survival.
Free Radic. Biol. Med., Apr;40(7):1108-18 (2006)
Enhancement of angiogenic effectors through hypoxia-inducible factor in preterm primate lung in vivo.
Am. J. Physiol. Lung Cell Mol. Physiol., Oct;291(4):L588-95 (2006)
Purinergic signaling and kinase activation for survival in pulmonary oxidative stress and disease.
Free Radic. Biol. Med., Jul;41(1):29-40 (2006)
Improved lung growth and function through hypoxia-inducible factor in primate chronic lung disease of prematurity.
FASEB J., Aug;20(10):1698-700 (2006)
Effect of cell cycle growth arrest on global DNA methylation status in human lung epithelial-like (A549) cells.
In Vivo., 20(6B):861-5 (2006)
Reduced thioredoxin increases proinflammatory cytokines and neutrophil influx in rat airways: modulation by airway mucus.
Free Radic. Biol. Med., May;42(9):1441-53 (2007)
Bcl-2 suppresses sarcoplasmic/endoplasmic reticulum Ca2+-ATPase expression in cystic fibrosis airways: role in oxidant-mediated cell death.
Am. J. Respir. Crit. Care Med., May;179(9):816-26 (2009)
Elevated expression of hexokinase II protects human lung epithelial-like A549 cells against oxidative injury.
Am. J. Physiol. Lung Cell Mol. Physiol., Sep;283(3):L573-84 (2002)
Extracellular ATP is an autocrine/paracrine regulator of hypoxia-induced adventitial fibroblast growth. Signaling through extracellular signal-regulated kinase-1/2 and the Egr-1 transcription factor.
J. Biol. Chem., Nov;277(47):44638-50 (2002)
Thioredoxin liquefies and decreases the viscoelasticity of cystic fibrosis sputum.
Am. J. Physiol. Lung Cell Mol. Physiol., May;286(5):L931-8 (2004)
Hyperoxia-induced DNA damage causes decreased DNA methylation in human lung epithelial-like A549 cells.
Antioxid. Redox Signal., Feb;6(1):129-36 (2004)