Partners healthcare system
Beth israel deaconess medical center
Brigham and womens hospital
Caritas holy family hospital and medical center
Caritas norwood hospital
Lawrence memorial hospital
Massachusetts eye and ear infirmary
Metrowest medical center
Morton hospital and medical center
Mount auburn hospital
Newton wellesley hospital
Rhode island hospital
St elizabeth medical center
Umass memorial medical center
Massachusetts General Hospital
Scavenger receptors in atherosclerosis: beyond lipid uptake.
Arterioscler. Thromb. Vasc. Biol., Aug;26(8):1702-11 (2006)
A sequence-oriented comparison of gene expression measurements across different hybridization-based technologies.
Nat. Biotechnol., Jul;24(7):832-40 (2006)
ABCA3 inactivation in mice causes respiratory failure, loss of pulmonary surfactant, and depletion of lung phosphatidylglycerol.
J. Lipid Res., Mar;48(3):621-32 (2007)
Targeting Innate Immunity for CV Benefit.
Drug Discov Today Ther Strateg., 5(1):15-23 (2008)
SPTLC1 binds ABCA1 to negatively regulate trafficking and cholesterol efflux activity of the transporter.
Biochemistry., Jun;47(23):6138-47 (2008)
Loss of SR-A and CD36 activity reduces atherosclerotic lesion complexity without abrogating foam cell formation in hyperlipidemic mice.
Arterioscler. Thromb. Vasc. Biol., Jan;29(1):19-26 (2009)
ABCA12 maintains the epidermal lipid permeability barrier by facilitating formation of ceramide linoleic esters.
J. Biol. Chem., Dec;283(52):36624-35 (2008)
Local injection of bevacizumab (Avastin) and angiolytic KTP laser treatment of recurrent respiratory papillomatosis of the vocal folds: a prospective study.
Ann. Otol. Rhinol. Laryngol., Oct;120(10):627-34 (2011)
The Creating an Optimal Warfarin Nomogram (CROWN) Study.
Thromb. Haemost., Jan;107(1):59-68 (2012)
A CD36-initiated signaling cascade mediates inflammatory effects of beta-amyloid.
J. Biol. Chem., Dec;277(49):47373-9 (2002)
Scavenger receptors class A-I/II and CD36 are the principal receptors responsible for the uptake of modified low density lipoprotein leading to lipid loading in macrophages.
J. Biol. Chem., Dec;277(51):49982-8 (2002)
CD36 mediates the innate host response to beta-amyloid.
J. Exp. Med., Jun;197(12):1657-66 (2003)
Activation of signaling pathways by putative scavenger receptor class A (SR-A) ligands requires CD14 but not SR-A.
Biochem. Biophys. Res. Commun., Oct;310(2):542-9 (2003)
Cross-linking and lipid efflux properties of apoA-I mutants suggest direct association between apoA-I helices and ABCA1.
Biochemistry., Feb;43(7):2126-39 (2004)
Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways.
Nat. Med., Apr;10(4):416-21 (2004)
ATP-binding cassette transporter A1 contains a novel C-terminal VFVNFA motif that is required for its cholesterol efflux and ApoA-I binding activities.
J. Biol. Chem., Nov;279(46):48477-85 (2004)
The induction of macrophage gene expression by LPS predominantly utilizes Myd88-independent signaling cascades.
Physiol. Genomics., Nov;19(3):319-30 (2004)
Abca7 null mice retain normal macrophage phosphatidylcholine and cholesterol efflux activity despite alterations in adipose mass and serum cholesterol levels.
J. Biol. Chem., Feb;280(5):3989-95 (2005)
Atherosclerosis and innate immune signaling.
Ann. Med., 37(2):130-40 (2005)
Inhibition of atherogenesis in BLT1-deficient mice reveals a role for LTB4 and BLT1 in smooth muscle cell recruitment.
Circulation., Jul;112(4):578-86 (2005)