Testosterone attenuates expression of vascular cell adhesion molecule-1 by conversion to estradiol by aromatase in endothelial cells: implications in atherosclerosis.
Proc. Natl. Acad. Sci. U.S.A., Mar;99(6):4055-60 (2002)
Caspase-8-mediated BID cleavage and release of mitochondrial cytochrome c during Nomega-hydroxy-L-arginine-induced apoptosis in MDA-MB-468 cells. Antagonistic effects of L-ornithine.
J. Biol. Chem., Oct;277(40):37630-6 (2002)
17-epiestriol, an estrogen metabolite, is more potent than estradiol in inhibiting vascular cell adhesion molecule 1 (VCAM-1) mRNA expression.
J. Biol. Chem., Apr;278(14):11746-52 (2003)
Nitric-oxide-induced Bax integration into the mitochondrial membrane commits MDA-MB-468 cells to apoptosis: essential role of Akt.
Cancer Res., Sep;63(17):5470-9 (2003)
MKP-1-induced dephosphorylation of extracellular signal-regulated kinase is essential for triggering nitric oxide-induced apoptosis in human breast cancer cell lines: implications in breast cancer.
Cancer Res., Dec;63(24):8853-60 (2003)
Serum levels of soluble vascular cell adhesion molecule-1 are decreased in women receiving oral contraceptives compared with normally menstruating women: implications in atherosclerosis.
Fertil. Steril., May;83(5):1480-8 (2005)
Nitric oxide, N omega-hydroxy-L-arginine and breast cancer.
Nitric Oxide., Sep;19(2):103-6 (2008)
Maintenance of higher H(2)O(2) levels, and its mechanism of action to induce growth in breast cancer cells: Important roles of bioactive catalase and PP2A.
Protein phosphatase 2A promotes endothelial survival via stabilization of translational inhibitor 4E-BP1 following exposure to tumor necrosis factor-Î±.
Arterioscler. Thromb. Vasc. Biol., Nov;31(11):2586-94 (2011)
Nitric oxide in physiologic concentrations targets the translational machinery to increase the proliferation of human breast cancer cells: involvement of mammalian target of rapamycin/eIF4E pathway.
Cancer Res., Jan;67(1):289-99 (2007)