Low dose estrogen prevents neuronal degeneration and microglial reactivity in an acute model of spinal cord injury: effect of dosing, route of administration, and therapy delay.
Neurochem. Res., Oct;36(10):1809-16 (2011)
Percutaneous cement augmentation of a lytic lesion of C1 via posterolateral approach under CT guidance.
Spine J., Jun;12(6):500-6 (2012)
Traumatic spinal cord injury mortality, 1981-1998.
J Trauma., Jan;66(1):184-90 (2009)
Cranial neuropathies after intracranial Photofrin-photodynamic therapy for malignant supratentorial gliomas-a report on 3 cases.
Surg Neurol., Aug;70(2):190-3 (2008)
Estrogen partially down-regulates PTEN to prevent apoptosis in VSC4.1 motoneurons following exposure to IFN-gamma.
Brain Res., Dec;1301:163-70 (2009)
The inhibition of apoptosis by melatonin in VSC4.1 motoneurons exposed to oxidative stress, glutamate excitotoxicity, or TNF-alpha toxicity involves membrane melatonin receptors.
J. Pineal Res., Mar;48(2):157-69 (2010)
Estrogen receptor agonists and estrogen attenuate TNF-Î±-induced apoptosis in VSC4.1 motoneurons.
J. Endocrinol., Feb;208(2):171-82 (2011)
Neuroprotective effects of genistein in VSC4.1 motoneurons exposed to activated microglial cytokines.
Neurochem. Int., Aug;59(2):175-84 (2011)
Overexpression of melatonin membrane receptors increases calcium-binding proteins and protects VSC4.1 motoneurons from glutamate toxicity through multiple mechanisms.